Module 5 Quiz

A  an underestimation of electrolyte concentration by indirect ISE methods when hyperproteinemia or hyperlipemia is present.

B  an overestimation of electrolyte concentration by direct ISE measurements only when serum is used.

C  the exclusion of certain solids by increased activity of electrolytes when determining concentration of those solids.

D  the exclusion of electrolytes in certain measurements when pH, PCO2, and PO2 are elevated in arterial blood.

A  shift to the right.

B  remain unchanged.

C  shift to the left.

A  the amount of total CO2 bound to hemoglobin.

B  the PO2 of alveolar air.

C  the hemoglobin binding capacity of blood.

A  Chloride

B  Sodium

C  Bicarbonate

A  moles of osmotic substances per liter

B  moles of osmotic substances per kilogram

C  moles per liter

D  equivalents per liter

A  oxygen dissociation.

B  oxygen saturation.

C  P50 is defined as the PO2 at which the hemoglobin of the blood is half saturated with O2.

D  P50.

E  the half-life of oxygen.

A  Lp(a) contains more core lipids, triglyceride, and cholesteryl ester and a smaller percent of protein.

B  Lp(a) contains a carbohydrate-rich protein called apo(a) that is covalently bound to apo B-100 through a disulfide linkage.

C  Lp(a) does not contain any apo B-100.

D  Lp(a) interacts with ABCA1 in peripheral cells, such as the macrophages, to remove additional cholesterol.

A  Cholesterol oxidase

B  HMG CoA reductase

C  Fatty acid hydrolase

D  Lecithin cholesterol acyltransferase

A  terpenes.

B  acylglycerols.

C  prostaglandins.

D  sphingolipids.

A  measurement of the rate of oxygen consumption occurs before removal of non-HDL lipoproteins.

B  precipitation of non-HDL lipoproteins must occur using polyanions.

C  enzymatic measurement of HDL occurs after non-HDL lipoproteins are removed by ultracentrifugation.

D  there is no physical separation of HDL from the non-HDL fractions and HDL is selectively measured.

A  Tangier disease

B  Insulin deficiency

C  Genetic lack of LDL receptors on cell surfaces

D  Lipoprotein lipase deficiency

A  Intracellular-cholesterol transport

B  Reverse-cholesterol transport

C  Endogenous

D  Exogenous

A  It is a myocardial protein that regulates muscle contraction through interaction with myosin and actin.

B  It is an enzyme found in skeletal muscle and cardiac muscle and is released upon tissue injury.

C  It is an acute phase reactant plasma protein that rises in response to inflammation, which has been demonstrated to be associated with risk of future CHD.

D  CRP levels rise because of its presence in cardiac muscle and release upon muscle cell death.

A  Self-monitoring method

B  Hexokinase method

C  Polarographic method

D  Glucose oxidase method

A  Glucose-6-phosphate and adenosine diphosphate (ADP)

B  Glycosylated hemoglobin

C  Nicotinamide adenine dinucleotide and NADH

D  6-Phosphogluconate and hydrogen ions

A  glycogen; the liver

B  glycoprotein; cell membranes

C  cellulose; vegetable fibers

D  starch; skeletal muscle

A  Glucagon inhibits gluconeogenesis.

B  Glucagon stimulates insulin release.

C  Glucagon inhibits glycogenesis.

D  Glucagon stimulates gluconeogenesis.

A  glycogenesis.

B  glycolysis.

C  glyconeogenesis.

D  glycogenolysis.

A  glycogenesis.

B  glycogenolysis.

C  glycolysis.

D  gluconeogenesis.

A  Postprandial glucose analysis

B  Assessment of fasting blood glucose during a hypoglycemic episode

C  Oral glucose tolerance test

D  A 72-hour fast