Exam 2

A  from top to bottom.

B  on the basis of wavelength.

C  retinotopically.

D  from left to right.

E  ipsilaterally.

A  K+ ions into neurons and Na+ ions out of neurons.

B  Na+ ions into neurons and K+ ions into neurons

C  Na+ ions into neurons

D  none of these

A  frontal lobes.

B  fusiform face area.

C  dorsal stream.

D  hippocampus.

A  small-molecule neurotransmitters.

B  acetylcholine.

C  glutamate.

D  neuropeptides.

E  dopamine.

A  color constancy.

B  the cocktail sausage demonstration.

C  the complementary color afterimage demonstration.

D  lateral inhibition.

E  the Mach band demonstration.

A    

B  PET

C  fMRI

D  MEG

E  CT

F  MRI

A  lordosis quotient

B  mount frequency

C  postejaculatory interval

D  number of ejaculations to intromission

E  number of intromissions to ejaculation

A  expulsion.

B  synthesis.

C  metabolism.

D  exocytosis.

E  excitation.

A  receptor layer, bipolar cell layer, retinal ganglion cell layer

B  none of these

C  retinal ganglion cell layer, horizontal cell layer, receptor layer and retinal ganglion cell layer, amacrine cell layer, receptor layer

D  receptor layer, bipolar cell layer, retinal ganglion cell layer and retinal ganglion cell layer, amacrine cell layer, receptor layer

A  angiography

B  MRI

C  CT

D  PET

E  EEG

A  rhodopsin.

B  the photopic system.

C  duplexity.

D  the scotopic system.

E  rods.

A  PET

B  ERP

C  TMS

D  EEG

E  2-DG

A  the dominant wavelength that it reflects.

B  none of these

C  all of these

D  its reflectance and the proportion of light of different wavelengths that it reflects.

A  complex cells or hypercomplex cells.

B  on-center or off-center cells.

C  type A or type B cells.

D  simple cells or complex cells.

E  hypercomplex cells.

A  all of these

B  triggers inhibitory effects.

C  closes rod sodium channels.

D  reduces the release of glutamate from rods.

E  hyperpolarizes the rods.

A  are more prevalent.

B  produce effects that take longer to develop.

C  all of these

D  produce longer lasting effects.

E  produce effects that are more diffuse.

A  anterior cingulate cortex.

B  inferotemporal cortex.

C  PAG.

D  secondary somatosensory cortex.

E  posterior parietal cortex.

A  CT scans

B  X-ray photography

C  PET scans

D  cerebral angiography

E  pneumoencephalography

A  indolamines

B  catecholamines

C  monoamines

D  amino acids

A  the damage that it produces lasts only a few weeks.

B  the damage that it produces lasts only a day or two.

C  the subjects usually survive.

D  it produces lesions that can be reversed with drugs.

E  it can temporarily suppress neural activity in a particular area of the brain without damaging the brain.

A  ERP

B  sodium amytal test.

C  dichotic listening test.

D  Psychiatric Test Inventory (PTI).

E  WAIS.

A  respond best to straight-line stimuli in a particular orientation.

B  have rectangular receptive fields.

C  respond to contrast.

D  are unresponsive to diffuse light.

E  all of these

A  nodes of Ranvier.

B  myelin.

C  ligand-activated ion channels.

D  EPSPs.

E  voltage-activated ion channels.

A  there is an IPSP.

B  the degree of depolarization on the axon adjacent to the hillock exceeds the threshold of excitation.

C  its sodium-potassium pumps are stimulated.

D  its buttons are stimulated.

E  there is an EPSP.

A  ionotropism.

B  metabotropism.

C  cohabitation.

D  covalence.

E  coexistence.

A  contrast.

B  straight lines.

C  dots of light.

D  circles.

E  movement.

A  the release of calcium ions from the buttons.

B  an influx of calcium ions.

C  an efflux of sodium ions.

D  the arrival of an AP at the axon hillock.

E  the sodium-potassium pump.

A  APs.

B  second messengers.

C  EPSPs.

D  IPSPs.

E  neurotransmitter.

A  PET

B  CT

C  functional MRI

D  EEG

E  angiography