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Fresh Grad Lands Job as Real Estate Agent With Help from Professional Writers

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Manush’s story shows the importance of using powerful keywords to his resume in landing the job he wanted.

CNS Pharmacology

Navigation   » List of Schools  »  West Coast University  »  Pharmacy  »  Phar 642 – Principles of Drug Action  »  Spring 2022  »  CNS Pharmacology

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Below are the questions for the exam with the choices of answers:

Question #1
A 33-year-old female patient was prescribed a drug for schizophrenia that increases dopamine release in the mesocortical pathway while decreasing dopamine release in the mesolimbic system. What drug was the patient most likely prescribed?
A  Haloperidol
B  Clozapine
C  Quetiapine
D  Risperidone
Question #2
What is an example of a drug that prevents the conversion of L-DOPA to 3-o-methyldopa in the periphery?
A  Rotigotine
B  Carbidopa
C  Entacapone
D  Selegiline
Question #3
In patients, what symptom is an indication of toxicity from L-DOPA?
A  Dyskinesia
B  Hypokinesia
C  Akinesia
D  Bradykinesia
Question #4
A 24-year-old male patient that had been taking haloperidol for schizophrenia presented with gynecomastia. Decreased dopamine activity in what brain region most likely contributed to this adverse effect of haloperidol?
A  Area postrema
B  Mesocortical pathway
C  Nigrostriatal pathway
D  Tuberoinfundibular pathway
E  Mesolimbic pathway
Question #5
What is the impact of dopamine loss on the direct and indirect pathways of the basal ganglia in patients with Parkinson disease?
A  Activity is decreased in the direct and indirect pathways
B  Activity is increased in the direct pathway and decreased in the indirect pathway
C  Activity is decreased in the direct pathway increased in the indirect pathway
D  Activity is increased in the direct and indirect pathways
Question #6
What dopamine receptors are located in the basal ganglia and in the area postrema?
A  D5
B  D4
C  D3
D  D2
Question #7
What enzyme converts L-DOPA to dopamine?
A  catechol-O-methyltransferase
B  Monoamine oxidase
C  Tyrosine hydroxylase
D  DOPA decarboxylase
Question #8
What is an example of a NMDA receptor antagonist used for Alzheimer disease?
A  Felbamate
B  Ketamine
C  Rufinamide
D  Memantine
Question #9
What are the main clinical uses of etomidate and propofol?
A  Anxiety
B  Seizures
C  Anesthesia
D  Insomnia
Question #10
What is the overall effect of flumazenil on the actions of GABA at GABA-A receptors?
A  It activates GABA receptors
B  It decreases GABA activity
C  It has no effect on GABA activity
D  It increases GABA activity
Question #11
What happens to glutamate receptors when a patient has Alzheimer disease?
A  Presynaptic autoreceptors become hyperactive
B  AMPA receptors become less sensitive
C  NMDA receptors become more sensitive
D  GABA receptors become dysfunctional
Question #12
What is a potential result of increasing GABAergic input in the thalamocortical neurons of a patient who experiences absence seizures?
A  No effect on absence seizures
B  Reduced frequency of seizures
C  Increased frequency of seizures
Question #13
What is a step necessary for the activation of AMPA receptors?
A  Displacement of magnesium
B  Glutamate binding
C  Prolonged intense stimulation
D  Co-activation with glycine
Question #14
What is the most direct consequence of activating a GABA-B receptor?
A  Decreased activation of adenylyl cyclase
B  Increased activity of protein kinase A
C  Influx of inhibitor ions
D  Influx of excitatory ions
Question #15
What effect would a drug that inhibits the activity of glutamic acid decarboxylase have on amino acid neurotransmitter levels?
A  Glutamate would increase and GABA would decrease
B  Glutamate would decrease and GABA would increase
C  Glutamate would increase and GABA would increase
D  Glutamate would decrease and GABA would decrease
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Fresh Graduate
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