Module 5 Quiz

A  the exclusion of certain solids by increased activity of electrolytes when determining concentration of those solids.

B  an overestimation of electrolyte concentration by direct ISE measurements only when serum is used.

C  an underestimation of electrolyte concentration by indirect ISE methods when hyperproteinemia or hyperlipemia is present.

D  the exclusion of electrolytes in certain measurements when pH, PCO2, and PO2 are elevated in arterial blood.

A  shift to the right.

B  shift to the left.

C  remain unchanged.

A  the hemoglobin binding capacity of blood.

B  the amount of total CO2 bound to hemoglobin.

C  the PO2 of alveolar air.

A  Sodium

B  Chloride

C  Bicarbonate

A  moles of osmotic substances per liter

B  moles of osmotic substances per kilogram

C  equivalents per liter

D  moles per liter

A  oxygen saturation.

B  oxygen dissociation.

C  P50.

D  P50 is defined as the PO2 at which the hemoglobin of the blood is half saturated with O2.

E  the half-life of oxygen.

A  Lp(a) interacts with ABCA1 in peripheral cells, such as the macrophages, to remove additional cholesterol.

B  Lp(a) does not contain any apo B-100.

C  Lp(a) contains a carbohydrate-rich protein called apo(a) that is covalently bound to apo B-100 through a disulfide linkage.

D  Lp(a) contains more core lipids, triglyceride, and cholesteryl ester and a smaller percent of protein.

A  Lecithin cholesterol acyltransferase

B  Fatty acid hydrolase

C  HMG CoA reductase

D  Cholesterol oxidase

A  sphingolipids.

B  prostaglandins.

C  acylglycerols.

D  terpenes.

A  there is no physical separation of HDL from the non-HDL fractions and HDL is selectively measured.

B  enzymatic measurement of HDL occurs after non-HDL lipoproteins are removed by ultracentrifugation.

C  measurement of the rate of oxygen consumption occurs before removal of non-HDL lipoproteins.

D  precipitation of non-HDL lipoproteins must occur using polyanions.

A  Insulin deficiency

B  Tangier disease

C  Lipoprotein lipase deficiency

D  Genetic lack of LDL receptors on cell surfaces

A  Endogenous

B  Exogenous

C  Intracellular-cholesterol transport

D  Reverse-cholesterol transport

A  It is an acute phase reactant plasma protein that rises in response to inflammation, which has been demonstrated to be associated with risk of future CHD.

B  It is an enzyme found in skeletal muscle and cardiac muscle and is released upon tissue injury.

C  CRP levels rise because of its presence in cardiac muscle and release upon muscle cell death.

D  It is a myocardial protein that regulates muscle contraction through interaction with myosin and actin.

A  Glucose oxidase method

B  Hexokinase method

C  Polarographic method

D  Self-monitoring method

A  Glucose-6-phosphate and adenosine diphosphate (ADP)

B  Nicotinamide adenine dinucleotide and NADH

C  Glycosylated hemoglobin

D  6-Phosphogluconate and hydrogen ions

A  glycogen; the liver

B  starch; skeletal muscle

C  cellulose; vegetable fibers

D  glycoprotein; cell membranes

A  Glucagon stimulates gluconeogenesis.

B  Glucagon stimulates insulin release.

C  Glucagon inhibits glycogenesis.

D  Glucagon inhibits gluconeogenesis.

A  glycogenesis.

B  glycogenolysis.

C  glyconeogenesis.

D  glycolysis.

A  gluconeogenesis.

B  glycogenolysis.

C  glycogenesis.

D  glycolysis.

A  A 72-hour fast

B  Postprandial glucose analysis

C  Assessment of fasting blood glucose during a hypoglycemic episode

D  Oral glucose tolerance test